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Course: Spiked Helmet Sign in the Inferior Leads

CME Credits: 1.00

Released: 2023-07-10

A patient in their 80s with a medical history of type 2 diabetes, hypertension, and poorly differentiated pulmonary adenocarcinoma was admitted to the critical care unit after experiencing a pulseless electrical activity cardiac arrest while receiving intravenous chemotherapy at an outpatient cancer center. They received 20 minutes of bystander chest compressions before the return of spontaneous circulation. On arrival to the intensive care unit, they were afebrile, their heart rate was 116 beats per minute, and their blood pressure was 85/55 mm Hg on mechanical ventilation. Laboratory examination results were unrevealing. Initial electrocardiogram (ECG) showed sinus tachycardia, heart rate of 109 beats per minute with normal intervals, and no acute ST-segment changes. Chest computed tomography angiography ruled out pulmonary embolism and aortic pathology. A transthoracic echocardiogram showed normal biventricular size and function without substantial valvular abnormalities. When intravenous norepinephrine was initiated, ST-segment and T-wave changes were noted on telemetry and confirmed on a 12-lead ECG, as shown in the , A.


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